Læknablaðið : fylgirit - 01.05.1978, Blaðsíða 122
antigens have been unequivocal. Grumet & al.
found an increase in HL-A8 and W 15, but
Kissmeyer-Nielsen found no such association in
a group of 65 Danish SLE-patients^).
As to the properdin factor B phenotype deter-
minations we can not say we have found an
increase in any special Bf-phenotypes. This
field of possible association between individual
Bf-phenotypes and disease does not seem to have
been much investigated so far. Quite different
from this, of course, is the recent interest in
properdin depositions in dermal and renal lesions
in S LE-patients 12,13).
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