Læknablaðið - 01.12.1971, Qupperneq 14

250
LÆKNABLAÐIÐ
TABLE 1
Deaths due to carbon monoxide poisoning.
Case nr. Year of death Age (years) Hb. Sat. with CO <%) Alcohol. Blood (%B) Conc. Urine (%c) Other drugs in blood
i 1966 38 71 1.02 0 b
2 1966 66 23 0.00 — 0 b
3 1967 65 50 2.37 — 0 b
4 1968 49 64 1.79 2.35 0 b, c, d, m
5 1968 35 79 1.77 3.85 0 b
6 1969 31 52 1.27 — 0 b, m
7 1969 41 60 2.70 3.84
8 1969 48 67 2.65 3.75
9 1969 38 88 2.70 4.45
10 1969 40 73 2.25 3.55
11 1969 38 76 3.37 4.35
12 1969 32 68 1.78 3.30
13 1969 47 90 1.50 1.56 0 b, c, d, m, n
14 1969 18 22 0.91 1.88
15 1969 21 54 1.04 2.12
16 1970 77 46 0.40 —
17 1970 24 58 2.12 2.72 0 b
18 1970 55 43 1.06 0.70 0 b
19 1970 27 48 1.96 3.46 0 b
Determinations of the degree of carbon monoxide saturation of
haemoglobin were performed by the method of Wolff (1941), in
modification. The concentration of alcohol in blood and urine was
determined by the alcohol dehydrogenase method (cf. Brink, Bonn-
ichsen & Theorell 1954) and the results expressed as g/1, i. e. %0
(w/v).
0: not found; — : not investigated.
b: barbituric acid derivaties; c: chlordiazepoxide; d: diazepam; m:
meprobamate; n: nitrazepam. — Investigations of the presence of
these drugs were by means of thin layer chromatography.